The non-invasive treatment for
angina and heart failure
For people with angina or heart failure, even simple activities - such as going to the mailbox or walking the dog - can be challenging.
If you are one of these people, take heart. There is a non-invasive treatment called EECP® Therapy that clinical experience has shown to be safe and to have benefit for the treatment of angina and heart failure. Approximately 80% of patients who complete the 35-hour course of EECP® Therapy experience significant symptom relief that may last up to three years.
EECP® Therapy is an outpatient treatment for angina and heart failure. Treatments are usually given for an hour each day, five days a week, for a total of 35 hours. During the treatment, you lie on a comfortable treatment table with large blood pressure-like cuffs wrapped around your legs and buttocks. These cuffs inflate and deflate at specific times between your heartbeats. A continuous electrocardiogram (ECG) is used to set the timing so the cuffs inflate while the heart is at rest when it normally gets its supply of blood and oxygen. The cuffs deflate at the end of that rest period, just before the next heartbeat. The special sensor applied to your finger checks the oxygen level in your blood and monitors the pressure waves created by the cuff inflations and deflations.
EECP® (Enhanced External Counterpulsation) Therapy and ECP (External Counterpulsation) Therapy are the terms which describe the non-invasive, complimentary or alternative treatment prescribed by physicians for patients suffering from the symptoms of angina, congestive heart failure and other cardiovascular diseases. Both EECP Therapy, a name which has been trademarked by Vasomedical Inc., Westbury, NY in the marketing of their treatment method and treatment devices they manufacturer and ECP Therapy, the generic term used to describe all other forms of external counterpulsation (including the ECP systems manufactured by PSK – Chongqing, China) , are based upon the proven hemodynamic principal of counterpulsation, which involves increasing blood flow (volume and pressure) to the heart during diastole (when the heart is at rest) and reducing the workload and oxygen demand the heart uses to pump blood throughout the body in systole.
In addition to the clinically proven hemodynamic principal of counterpulsation, extensive research in clinical studies published in peer-reviewed medical journals have demonstrated the many mechanisms of action that are responsible for the initial benefits produced by the therapy as well as the long term outcomes, which show these initial benefits lasting three to five years. While every year a number of clinical articles on EECP and ECP continue to be published in medical journals and textbooks worldwide, some using PSK ECP systems and supported by PSK, most of the over 200 clinical articles published in the literature on EECP and ECP therapy were performed on Vasomedical EECP therapy systems and reported in the clinical research supported by Vasomedical.
Decisive advantage at a glance:
EECP®, is a registered trademark of Vasomedical and may only be used to describe the external counterpulsation therapy delivered by using a Vasomedical EECP® Therapy system. EECP® therapy has been clinically proven to be the highest standard of external counterpulsation. There are over 200 publications in medical journals, many of them peer-reviewed, demonstrating the clinical efficacy of EECP® therapy. This is the gold standard of clinical proof, in external counterpulsation.
The successful application of external counterpulsation involves several key elements:
#1. The Residual Pressure left in the cuff from the previous inflation/deflation cycle
#2. The Peak Pressure in each of the individual cuffs and the pressure gradient between cuffs.
#3. The Rise time for all three cuffs to reach Peak Pressure
#4. The duration of time that each of the individual cuffs is inflated at Peak Pressure.
#5. The Decay Time (deflation) for all three cuffs
#6. The uniformity and delivery of cuff pressure to the vascular bed.
These elements, properly applied, are necessary for the optimal increase in diastolic augmentation and upon deflation, provides for the maximal reduction of cardiac workload. This combined effect yields the best therapeutic counterpulsation.
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